 Receptor, Parathyroid Hormone, Type 1
"Receptor, Parathyroid Hormone, Type 1" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A parathyroid hormone receptor subtype that recognizes both PARATHYROID HORMONE and PARATHYROID HORMONE-RELATED PROTEIN. It is a G-protein-coupled receptor that is expressed at high levels in BONE and in KIDNEY.
| Descriptor ID |
D044168
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| MeSH Number(s) |
D12.776.543.750.100.650.100 D12.776.543.750.750.600.100
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| Concept/Terms |
Receptor, Parathyroid Hormone, Type 1- Receptor, Parathyroid Hormone, Type 1
- Parathyroid Hormone-Related Peptide Receptor
- Parathyroid Hormone Related Peptide Receptor
- PTH-PTHrP Receptor
- PTH PTHrP Receptor
- Receptor, PTH-PTHrP
- PTH-Related Peptide Receptor
- PTH Related Peptide Receptor
- Receptor, PTH-Related Peptide
- PTH-Related Protein Receptor
- PTH Related Protein Receptor
- Receptor, PTH-Related Protein
- PTHLP Receptor
- Receptor, PTHLP
- Parathyroid Hormone-Like Peptide Receptor
- Parathyroid Hormone Like Peptide Receptor
- Parathyroid Hormone Receptor
- Receptor, Parathyroid Hormone
- Parathyroid Hormone Receptor 1
- Parathyroid Hormone Receptor Type 1
- Parathyroid Hormone Receptor Type I
- Receptor, Parathyroid Hormone-Like Peptide
- PTHrP Receptor
- Receptor, PTHrP
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Below are MeSH descriptors whose meaning is more general than "Receptor, Parathyroid Hormone, Type 1".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Membrane Proteins [D12.776.543]
- Receptors, Cell Surface [D12.776.543.750]
- Receptors, G-Protein-Coupled [D12.776.543.750.100]
- Receptors, Parathyroid Hormone [D12.776.543.750.100.650]
- Receptor, Parathyroid Hormone, Type 1 [D12.776.543.750.100.650.100]
- Receptors, Peptide [D12.776.543.750.750]
- Receptors, Parathyroid Hormone [D12.776.543.750.750.600]
- Receptor, Parathyroid Hormone, Type 1 [D12.776.543.750.750.600.100]
Below are MeSH descriptors whose meaning is more specific than "Receptor, Parathyroid Hormone, Type 1".
This graph shows the total number of publications written about "Receptor, Parathyroid Hormone, Type 1" by people in UAMS Profiles by year, and whether "Receptor, Parathyroid Hormone, Type 1" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2024 | 1 | 0 | 1 | | 2022 | 0 | 1 | 1 | | 2018 | 0 | 1 | 1 | | 2017 | 0 | 1 | 1 | | 2016 | 1 | 0 | 1 | | 2013 | 2 | 0 | 2 | | 2012 | 1 | 0 | 1 | | 2011 | 2 | 0 | 2 | | 2008 | 1 | 0 | 1 | | 2006 | 0 | 1 | 1 | | 2005 | 1 | 0 | 1 | | 2002 | 0 | 1 | 1 |
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Below are the most recent publications written about "Receptor, Parathyroid Hormone, Type 1" by people in Profiles over the past ten years.
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Marino S, Bellido T. PTH receptor signalling, osteocytes and bone disease induced by diabetes mellitus. Nat Rev Endocrinol. 2024 Nov; 20(11):661-672.
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Delgado-Calle J, McAndrews K, Wu G, Orr AL, Ferrari A, Tu X, Srinivasan V, Roodman GD, Ebetino FH, Boeckman RK, Bellido T. The Notch pathway regulates the bone gain induced by PTH anabolic signaling. FASEB J. 2022 03; 36(3):e22196.
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Delgado-Calle J, Hancock B, Likine EF, Sato AY, McAndrews K, Sanudo C, Bruzzaniti A, Riancho JA, Tonra JR, Bellido T. MMP14 is a novel target of PTH signaling in osteocytes that controls resorption by regulating soluble RANKL production. FASEB J. 2018 05; 32(5):2878-2890.
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Yang Y, Lei H, Qiang YW, Wang B. Ixazomib enhances parathyroid hormone-induced ?-catenin/T-cell factor signaling by dissociating ?-catenin from the parathyroid hormone receptor. Mol Biol Cell. 2017 Jul 01; 28(13):1792-1803.
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Delgado-Calle J, Tu X, Pacheco-Costa R, McAndrews K, Edwards R, Pellegrini GG, Kuhlenschmidt K, Olivos N, Robling A, Peacock M, Plotkin LI, Bellido T. Control of Bone Anabolism in Response to Mechanical Loading and PTH by Distinct Mechanisms Downstream of the PTH Receptor. J Bone Miner Res. 2017 Mar; 32(3):522-535.
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