Lecturer, Graduate Pharmacology and Therapeutics (Three lectures)
1. Pharmacology of hematopoiesis and immunomodulation
2. Agents in diabetes
3. Adrenocorticosteroids and agonists

Lecturer, Pathologic Basis of Disease (Two lectures)
1. Endocrine disease – Diabetes
2. Endocrine disease – Diabetes

Lecturer, Principles and Methods of Pharmacology & Toxicology (One lecture)
Animal Models of Hypertension

Lecturer, Cell signaling (one lecture)
Multiple additional signaling mechanisms

Metabolic syndrome persists as a leading cause of morbidity and mortality among adults in the United States. Specifically, the co-existence of diabetes and hypertension, hallmark components of metabolic syndrome, stand as primary contributors to cardiovascular disease and subsequent mortality. Thus, it is important to identify the pathogenic connection between diabetes and hypertension. Ample evidence suggested the involvement of immune cells, particularly T cells, in the pathogenesis of diabetes and hypertension. However, the precise mechanisms by which immune dysregulation associated with diabetes contributes to hypertension are not fully understood. Our research centers on investigating new intrinsic cellular mechanisms existing in diabetic conditions, which sustain chronic T cell activation and accentuate the progression of cardiovascular complications.

The links below are provided by the researcher to provide access to external online bibliographies that they keep independently from the Profiles System.