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Search Results to Mitchell Mcgill

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Mcgill, Mitchell

One or more keywords matched the following items that are connected to Mcgill, Mitchell

Item Type	Name
Concept	Receptor-Interacting Protein Serine-Threonine Kinases
Concept	MAP Kinase Kinase 4
Concept	JNK Mitogen-Activated Protein Kinases
Concept	TOR Serine-Threonine Kinases
Academic Article	Lipin deactivation after acetaminophen overdose causes phosphatidic acid accumulation in liver and plasma in mice and humans and enhances liver regeneration.
Academic Article	Acetaminophen-induced liver injury in rats and mice: comparison of protein adducts, mitochondrial dysfunction, and oxidative stress in the mechanism of toxicity.
Academic Article	Low Dose Acetaminophen Induces Reversible Mitochondrial Dysfunction Associated with Transient c-Jun N-Terminal Kinase Activation in Mouse Liver.
Academic Article	Differences in early acetaminophen hepatotoxicity between obese ob/ob and db/db mice.
Academic Article	Purinergic receptor antagonist A438079 protects against acetaminophen-induced liver injury by inhibiting p450 isoenzymes, not by inflammasome activation.
Academic Article	Mechanisms of acetaminophen-induced cell death in primary human hepatocytes.
Academic Article	The role of the c-Jun N-terminal kinases 1/2 and receptor-interacting protein kinase 3 in furosemide-induced liver injury.
Academic Article	Resveratrol prevents protein nitration and release of endonucleases from mitochondria during acetaminophen hepatotoxicity.
Academic Article	Lower susceptibility of female mice to acetaminophen hepatotoxicity: Role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase.
Academic Article	Pathophysiological significance of c-jun N-terminal kinase in acetaminophen hepatotoxicity.
Academic Article	Bile Acid-Induced Toxicity in HepaRG Cells Recapitulates the Response in Primary Human Hepatocytes.
Academic Article	Receptor interacting protein kinase 3 is a critical early mediator of acetaminophen-induced hepatocyte necrosis in mice.
Academic Article	The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal kinase activation.
Academic Article	Mitochondrial protein adducts formation and mitochondrial dysfunction during N-acetyl-m-aminophenol (AMAP)-induced hepatotoxicity in primary human hepatocytes.
Academic Article	Protection against acetaminophen-induced liver injury by allopurinol is dependent on aldehyde oxidase-mediated liver preconditioning.
Academic Article	The inhibitor of glycerol 3-phosphate acyltransferase FSG67 blunts liver regeneration after acetaminophen overdose by altering GSK3? and Wnt/?-catenin signaling.

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Phosphotransferases