 Superoxides
"Superoxides" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Highly reactive compounds produced when oxygen is reduced by a single electron. In biological systems, they may be generated during the normal catalytic function of a number of enzymes and during the oxidation of hemoglobin to METHEMOGLOBIN. In living organisms, SUPEROXIDE DISMUTASE protects the cell from the deleterious effects of superoxides.
| Descriptor ID |
D013481
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| MeSH Number(s) |
D01.248.497.158.685.750.850 D01.339.431.374.850 D01.339.431.750 D01.650.550.750.800
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| Concept/Terms |
Superoxides- Superoxides
- Superoxide Radical
- Superoxide Anion
- Superoxide
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Below are MeSH descriptors whose meaning is more general than "Superoxides".
Below are MeSH descriptors whose meaning is more specific than "Superoxides".
This graph shows the total number of publications written about "Superoxides" by people in UAMS Profiles by year, and whether "Superoxides" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2023 | 0 | 1 | 1 | | 2022 | 0 | 1 | 1 | | 2019 | 0 | 1 | 1 | | 2018 | 0 | 2 | 2 | | 2017 | 0 | 1 | 1 | | 2016 | 2 | 0 | 2 | | 2015 | 0 | 1 | 1 | | 2014 | 0 | 1 | 1 | | 2013 | 3 | 1 | 4 | | 2012 | 1 | 1 | 2 | | 2011 | 2 | 0 | 2 | | 2010 | 1 | 4 | 5 | | 2009 | 2 | 0 | 2 | | 2008 | 0 | 1 | 1 | | 2007 | 0 | 1 | 1 | | 2006 | 0 | 3 | 3 | | 2005 | 1 | 3 | 4 | | 2004 | 1 | 4 | 5 | | 2003 | 0 | 2 | 2 | | 2002 | 0 | 3 | 3 | | 2001 | 0 | 1 | 1 | | 1999 | 0 | 2 | 2 | | 1998 | 0 | 1 | 1 | | 1997 | 1 | 1 | 2 | | 1995 | 0 | 1 | 1 | | 1992 | 0 | 1 | 1 | | 1991 | 0 | 1 | 1 |
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Below are the most recent publications written about "Superoxides" by people in Profiles over the past ten years.
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Meister ML, Feresin RG. Blackberry consumption protects against e-cigarette-induced vascular oxidative stress in mice. Food Funct. 2023 Dec 11; 14(24):10709-10730.
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Raghavan S, Kundumani-Sridharan V, Kumar S, White CW, Das KC. Thioredoxin Prevents Loss of UCP2 in Hyperoxia via MKK4-p38 MAPK-PGC1a Signaling and Limits Oxygen Toxicity. Am J Respir Cell Mol Biol. 2022 03; 66(3):323-336.
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Kundumani-Sridharan V, Subramani J, Raghavan S, Maiti GP, Owens C, Walker T, Wasnick J, Idell S, Das KC. Short-duration hyperoxia causes genotoxicity in mouse lungs: protection by volatile anesthetic isoflurane. Am J Physiol Lung Cell Mol Physiol. 2019 05 01; 316(5):L903-L917.
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Wongsurawat T, Woo CC, Giannakakis A, Lin XY, Cheow ESH, Lee CN, Richards M, Sze SK, Nookaew I, Kuznetsov VA, Sorokin V. Distinctive molecular signature and activated signaling pathways in aortic smooth muscle cells of patients with myocardial infarction. Atherosclerosis. 2018 04; 271:237-244.
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Ogunbileje JO, Herndon DN, Murton AJ, Porter C. The Role of Mitochondrial Stress in Muscle Wasting Following Severe Burn Trauma. J Burn Care Res. 2018 01 01; 39(1):100-108.
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Hilgers RH, Kundumani-Sridharan V, Subramani J, Chen LC, Cuello LG, Rusch NJ, Das KC. Thioredoxin reverses age-related hypertension by chronically improving vascular redox and restoring eNOS function. Sci Transl Med. 2017 02 08; 9(376).
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Imig JD. Renal blood flow autoregulation: what are the contributions for nitric oxide or superoxide to modulate the myogenic response? Am J Physiol Renal Physiol. 2016 05 15; 310(10):F1013-5.
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Stec DE, Juncos LA, Granger JP. Renal intramedullary infusion of tempol normalizes the blood pressure?response to intrarenal blockade of heme oxygenase-1 in?angiotensin II-dependent hypertension. J Am Soc Hypertens. 2016 Apr; 10(4):346-51.
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Falc?o SA, Weinkopff T, Hurrell BP, Celes FS, Curvelo RP, Prates DB, Barral A, Borges VM, Tacchini-Cottier F, de Oliveira CI. Exposure to Leishmania braziliensis triggers neutrophil activation and apoptosis. PLoS Negl Trop Dis. 2015 Mar; 9(3):e0003601.
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Kang L, Dai C, Lustig ME, Bonner JS, Mayes WH, Mokshagundam S, James FD, Thompson CS, Lin CT, Perry CG, Anderson EJ, Neufer PD, Wasserman DH, Powers AC. Heterozygous SOD2 deletion impairs glucose-stimulated insulin secretion, but not insulin action, in high-fat-fed mice. Diabetes. 2014 Nov; 63(11):3699-710.
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