 Vacuolar Proton-Translocating ATPases
"Vacuolar Proton-Translocating ATPases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proton-translocating ATPases that are involved in acidification of a variety of intracellular compartments.
| Descriptor ID |
D025262
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| MeSH Number(s) |
D08.811.277.040.025.325.875 D08.811.913.696.650.150.500.875 D12.776.157.530.450.250.875.500.875 D12.776.543.585.450.250.875.500.875
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| Concept/Terms |
Vacuolar Proton-Translocating ATPases- Vacuolar Proton-Translocating ATPases
- Lysosomal Proton-Translocating ATPases
- ATPases, Lysosomal Proton-Translocating
- Lysosomal Proton Translocating ATPases
- Proton-Translocating ATPases, Lysosomal
- V-Type ATPase
- ATPase, V-Type
- V Type ATPase
- Vacuolar ATPase
- ATPase, Vacuolar
- Vacuolar F(1)F(0) ATPases
- Vacuolar H+-ATPase
- H+-ATPase, Vacuolar
- Vacuolar H+ ATPase
- Vacuolar Membrane H(+)-ATPase
- Lysosomal F(1)F(0) ATPase
- Vacuolar F(1)F(0) ATPase
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Below are MeSH descriptors whose meaning is more general than "Vacuolar Proton-Translocating ATPases".
Below are MeSH descriptors whose meaning is more specific than "Vacuolar Proton-Translocating ATPases".
This graph shows the total number of publications written about "Vacuolar Proton-Translocating ATPases" by people in UAMS Profiles by year, and whether "Vacuolar Proton-Translocating ATPases" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2022 | 1 | 0 | 1 | | 2021 | 0 | 1 | 1 | | 2015 | 1 | 0 | 1 | | 2002 | 1 | 0 | 1 |
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Below are the most recent publications written about "Vacuolar Proton-Translocating ATPases" by people in Profiles over the past ten years.
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Edelman WC, Kiianitsa K, Virmani T, Martinez RA, Young JE, Keene CD, Bird TD, Raskind WH, Korvatska O. Reduced gene dosage is a common mechanism of neuropathologies caused by ATP6AP2 splicing mutations. Parkinsonism Relat Disord. 2022 08; 101:31-38.
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Hussein NA, Malla S, Pasternak MA, Terrero D, Brown NG, Ashby CR, Assaraf YG, Chen ZS, Tiwari AK. The role of endolysosomal trafficking in anticancer drug resistance. Drug Resist Updat. 2021 07; 57:100769.
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Seu KG, Trump LR, Emberesh S, Lorsbach RB, Johnson C, Meznarich J, Underhill HR, Chou ST, Sakthivel H, Nassar NN, Seu KJ, Blanc L, Zhang W, Lutzko CM, Kalfa TA. VPS4A Mutations in Humans Cause Syndromic Congenital Dyserythropoietic Anemia due to Cytokinesis and Trafficking Defects. Am J Hum Genet. 2020 12 03; 107(6):1149-1156.
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Gupta HV, Vengoechea J, Sahaya K, Virmani T. A splice site mutation in ATP6AP2 causes X-linked intellectual disability, epilepsy, and parkinsonism. Parkinsonism Relat Disord. 2015 Dec; 21(12):1473-5.
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