Phosphatidic Acids
"Phosphatidic Acids" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Fatty acid derivatives of glycerophosphates. They are composed of glycerol bound in ester linkage with 1 mole of phosphoric acid at the terminal 3-hydroxyl group and with 2 moles of fatty acids at the other two hydroxyl groups.
| Descriptor ID |
D010712
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| MeSH Number(s) |
D10.570.755.375.760
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| Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Phosphatidic Acids".
Below are MeSH descriptors whose meaning is more specific than "Phosphatidic Acids".
This graph shows the total number of publications written about "Phosphatidic Acids" by people in UAMS Profiles by year, and whether "Phosphatidic Acids" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2020 | 0 | 1 | 1 | | 2019 | 1 | 1 | 2 | | 2018 | 1 | 0 | 1 | | 2010 | 1 | 0 | 1 | | 1991 | 0 | 1 | 1 |
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Below are the most recent publications written about "Phosphatidic Acids" by people in Profiles over the past ten years.
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Onono FO, Morris AJ. Phospholipase D and Choline Metabolism. Handb Exp Pharmacol. 2020; 259:205-218.
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Park SY, Yang JS, Li Z, Deng P, Zhu X, Young D, Ericsson M, Andringa RLH, Minnaard AJ, Zhu C, Sun F, Moody DB, Morris AJ, Fan J, Hsu VW. The late stage of COPI vesicle fission requires shorter forms of phosphatidic acid and diacylglycerol. Nat Commun. 2019 07 30; 10(1):3409.
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Clemens MM, Kennon-McGill S, Apte U, James LP, Finck BN, McGill MR. The inhibitor of glycerol 3-phosphate acyltransferase FSG67 blunts liver regeneration after acetaminophen overdose by altering GSK3? and Wnt/?-catenin signaling. Food Chem Toxicol. 2019 Mar; 125:279-288.
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Lutkewitte AJ, Schweitzer GG, Kennon-McGill S, Clemens MM, James LP, Jaeschke H, Finck BN, McGill MR. Lipin deactivation after acetaminophen overdose causes phosphatidic acid accumulation in liver and plasma in mice and humans and enhances liver regeneration. Food Chem Toxicol. 2018 May; 115:273-283.
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