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Search Results to Yuet-Kin Leung

This is a "connection" page, showing the details of why an item matched the keywords from your search.

                     
                     

One or more keywords matched the following items that are connected to Leung, Yuet-Kin

Item TypeName
Concept Pyruvate Kinase
Concept MAP Kinase Signaling System
Concept Receptor Protein-Tyrosine Kinases
Concept fms-Like Tyrosine Kinase 3
Concept Extracellular Signal-Regulated MAP Kinases
Concept Protein Kinase Inhibitors
Concept p38 Mitogen-Activated Protein Kinases
Academic Article Phosphorylation of human estrogen receptor-beta at serine 105 inhibits breast cancer cell migration and invasion.
Academic Article ICI 182,780-regulated gene expression in DU145 prostate cancer cells is mediated by estrogen receptor-beta/NFkappaB crosstalk.
Academic Article Estrogen activates pyruvate kinase M2 and increases the growth of TSC2-deficient cells.
Academic Article Pyrrolo[2,3-d]pyrimidine derivatives as inhibitors of RET: Design, synthesis and biological evaluation.
Academic Article Discovery and biological evaluation of phthalazines as novel non-kinase TGF? pathway inhibitors.
Academic Article Discovery of 4-aminoquinolines as highly selective TGF?R1 inhibitors with an attenuated MAP4K4 profile for potential applications in immuno-oncology.
Academic Article Discovery of imidazo[1,2-a]pyridine-thiophene derivatives as FLT3 and FLT3 mutants inhibitors for acute myeloid leukemia through structure-based optimization of an NEK2 inhibitor.
Academic Article Discovery of N-Trisubstituted Pyrimidine Derivatives as Type I RET and RET Gatekeeper Mutant Inhibitors with a Novel Kinase Binding Pose.
Academic Article N-(3-Methoxyphenyl)-6-(7-(1-methyl-1H-pyrazol-4-yl)imidazo[1,2-a]pyridin-3-yl)pyridin-2-amine is an inhibitor of the FLT3-ITD and BCR-ABL pathways, and potently inhibits FLT3-ITD/D835Y and FLT3-ITD/F691L secondary mutants.
Academic Article An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L.

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  • Phosphotransferases