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Search Results to Mitchell Mcgill

This is a "connection" page, showing the details of why an item matched the keywords from your search.

                     
                     

One or more keywords matched the following items that are connected to Mcgill, Mitchell

Item TypeName
Concept Necrosis
Concept Tumor Necrosis Factor-alpha
Academic Article Plasma biomarkers to study mechanisms of liver injury in patients with hypoxic hepatitis.
Academic Article Caspase Inhibition Prevents Tumor Necrosis Factor-a-Induced Apoptosis and Promotes Necrotic Cell Death in Mouse Hepatocytes in?Vivo and in?Vitro.
Academic Article Mechanisms of acetaminophen-induced cell death in primary human hepatocytes.
Academic Article The mechanism underlying acetaminophen-induced hepatotoxicity in humans and mice involves mitochondrial damage and nuclear DNA fragmentation.
Academic Article HepaRG cells: a human model to study mechanisms of acetaminophen hepatotoxicity.
Academic Article Apoptosis or necrosis in acetaminophen-induced acute liver failure? New insights from mechanistic biomarkers*.
Academic Article RETRACTED: Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity.
Academic Article Receptor interacting protein kinase 3 is a critical early mediator of acetaminophen-induced hepatocyte necrosis in mice.
Academic Article Oxidant stress, mitochondria, and cell death mechanisms in drug-induced liver injury: lessons learned from acetaminophen hepatotoxicity.
Academic Article Current issues with acetaminophen hepatotoxicity--a clinically relevant model to test the efficacy of natural products.
Academic Article The inhibitor of glycerol 3-phosphate acyltransferase FSG67 blunts liver regeneration after acetaminophen overdose by altering GSK3? and Wnt/?-catenin signaling.
Academic Article Propagation of Pericentral Necrosis During Acetaminophen-Induced Liver Injury: Evidence for Early Interhepatocyte Communication and Information Exchange.
Academic Article Contrasting model mechanisms of alanine aminotransferase (ALT) release from damaged and necrotic hepatocytes as an example of general biomarker mechanisms.

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  • Necrosis