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Search Results to Mitchell Mcgill

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Mcgill, Mitchell

One or more keywords matched the following items that are connected to Mcgill, Mitchell

Item Type	Name
Concept	Hepatocytes
Academic Article	Removal of acetaminophen protein adducts by autophagy protects against acetaminophen-induced liver injury in mice.
Academic Article	Involvement of connexin43 in acetaminophen-induced liver injury.
Academic Article	Caspase Inhibition Prevents Tumor Necrosis Factor-a-Induced Apoptosis and Promotes Necrotic Cell Death in Mouse Hepatocytes in?Vivo and in?Vitro.
Academic Article	Cytochrome P450-derived versus mitochondrial oxidant stress in acetaminophen hepatotoxicity.
Academic Article	Lack of Direct Cytotoxicity of Extracellular ATP against Hepatocytes: Role in the Mechanism of Acetaminophen Hepatotoxicity.
Academic Article	Purinergic receptor antagonist A438079 protects against acetaminophen-induced liver injury by inhibiting p450 isoenzymes, not by inflammasome activation.
Academic Article	Mechanisms of acetaminophen-induced cell death in primary human hepatocytes.
Academic Article	Resveratrol prevents protein nitration and release of endonucleases from mitochondria during acetaminophen hepatotoxicity.
Academic Article	Plasma and liver acetaminophen-protein adduct levels in mice after acetaminophen treatment: dose-response, mechanisms, and clinical implications.
Academic Article	A cellular model to study drug-induced liver injury in nonalcoholic fatty liver disease: Application to acetaminophen.
Academic Article	Lysosomal instability and cathepsin B release during acetaminophen hepatotoxicity.
Academic Article	Pathophysiological significance of c-jun N-terminal kinase in acetaminophen hepatotoxicity.
Academic Article	Pathophysiological relevance of proteomics investigations of drug-induced hepatotoxicity in HepG2 cells.
Academic Article	Bile Acid-Induced Toxicity in HepaRG Cells Recapitulates the Response in Primary Human Hepatocytes.
Academic Article	Platelets and protease-activated receptor-4 contribute to acetaminophen-induced liver injury in mice.
Academic Article	Receptor interacting protein kinase 3 is a critical early mediator of acetaminophen-induced hepatocyte necrosis in mice.
Academic Article	The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal kinase activation.
Academic Article	Dual Role of Epidermal Growth Factor Receptor in Liver Injury and Regeneration after Acetaminophen Overdose in Mice.
Academic Article	Mitochondrial protein adducts formation and mitochondrial dysfunction during N-acetyl-m-aminophenol (AMAP)-induced hepatotoxicity in primary human hepatocytes.
Academic Article	Benzyl alcohol protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes but causes mitochondrial dysfunction and cell death at higher doses.
Academic Article	Propagation of Pericentral Necrosis During Acetaminophen-Induced Liver Injury: Evidence for Early Interhepatocyte Communication and Information Exchange.
Academic Article	Lack of direct cytotoxicity of extracellular ATP against hepatocytes: role in the mechanism of acetaminophen hepatotoxicity.
Academic Article	Hepatotoxicity of a Cannabidiol-Rich Cannabis Extract in the Mouse Model.
Academic Article	Contrasting model mechanisms of alanine aminotransferase (ALT) release from damaged and necrotic hepatocytes as an example of general biomarker mechanisms.

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Hepatocytes