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Search Results to Robert Weinstein

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Weinstein, Robert

One or more keywords matched the following items that are connected to Weinstein, Robert

Item Type	Name
Academic Article	Glucocorticoids act directly on osteoblasts and osteocytes to induce their apoptosis and reduce bone formation and strength.
Academic Article	Quantifying osteoblast and osteocyte apoptosis: challenges and rewards.
Academic Article	Glucocorticoids, osteocytes, and skeletal fragility: the role of bone vascularity.
Academic Article	Intermittent parathyroid hormone administration counteracts the adverse effects of glucocorticoids on osteoblast and osteocyte viability, bone formation, and strength in mice.
Academic Article	Connexin 43 is required for the anti-apoptotic effect of bisphosphonates on osteocytes and osteoblasts in vivo.
Academic Article	Osteocyte apoptosis.
Academic Article	Osteoprotegerin prevents glucocorticoid-induced osteocyte apoptosis in mice.
Academic Article	Glucocorticoid-induced osteoporosis and osteonecrosis.
Academic Article	Control of bone mass and remodeling by PTH receptor signaling in osteocytes.
Academic Article	Suppression of autophagy in osteocytes mimics skeletal aging.
Academic Article	Osteocyte apoptosis is induced by weightlessness in mice and precedes osteoclast recruitment and bone loss.
Academic Article	Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age.
Academic Article	Glucocorticoid-induced osteonecrosis.
Concept	Osteocytes
Academic Article	Preservation of osteocyte viability by bisphosphonates contributes to bone strength in glucocorticoid-treated mice independently of BMD: an unappreciated determinant of bone strength
Academic Article	Intermittent parathyroid hormone administration prevents glucocorticoid-induced osteoblast and osteocyte apoptosis, decreased bone formation, and reduced bone strength in mice
Academic Article	Prednisolone Administration Ameliorates the Increased Osteoclastogenesis But Aggravates the Loss of Osteocytes and Bone Strength That Follow Orchidectomy
Academic Article	Osteoprotegerin prevents glucocorticoid-induced osteocyte apoptosis, decreased bone interstitial fluid, and reduced strenght in mice( in preparation
Academic Article	Glucocorticoid excess causes apoptosis of osteocytes in murine cortical bone
Academic Article	Dysfunctional Osteocytes Increase RANKL and Promote Cortical Pore Formation in Their Vicinity: a Mechanical Explanation for the Development of Cortical Porosity with Age
Academic Article	Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates bone porosity with age
Academic Article	Like Estrogen, Androgen Exert Potent and Direct Anti-apoptotic Effects on Osteoblasts and Osteocytes in vivo and in vitro
Academic Article	Review-Glucocorticoids, Osteocytes and Skeletal fragilty: The Role of Bone Vascularity
Academic Article	Anatomic juxtaposition of apoptotic osteocytes and avascular necrosis in femurs from patients with glucocorticoid excess
Academic Article	Glucocorticoids Act Directly on Osteocytes to Reduce Bone Vascularity and Strength
Academic Article	Suppression of Autophagy in Osteoblasts and Osteocytes Increases Oxidative Stress and Recapitulates the Effects of Aging on the Murine Skeleton
Academic Article	Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency.
Academic Article	Suppression of autophagy in osteocytes does not modify the adverse effects of glucocorticoids on cortical bone.
Grant	Glucocorticoid-induced osteonecrosis of the hip, osteocytes and canalicular fluid
Grant	Molecular and Cellular Mechanisms of Osteoporosis
Grant	Osteocyte Control of Bone Remodeling
Grant	The Role of Osteocyte Survival in Bone Strength
Grant	Bisphosphonate effects on osteoclasts and osteocytes in vitro: mechanisms of inhibition of glucocorticoid-induced apoptosis of osteocytes
Grant	Bisphosphonate effects on osteoclasts and osteocytes in glucocorticoid-induced osteoporosis: relationship to fracture prevention.
Grant	Bisphosphonate effects on osteoclasts and osteocytes in vitro: mechanisms of inhibition of glucocorticoid-induced apoptosis of osteocytes
Grant	Bisphosphonate effects on osteoclasts and osteocytes in glucocorticoid-induced osteoporosis: relationship to fracture prevention.

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Osteocytes