Connection

Announcement

You can now add alternative names! Click here to add other names that you've published under.

Search Results to Robert Weinstein

This is a "connection" page, showing the details of why an item matched the keywords from your search.

Search Results

Weinstein, Robert

One or more keywords matched the following items that are connected to Weinstein, Robert

Item Type	Name
Academic Article	The skeletal effects of glucocorticoid excess override those of orchidectomy in mice.
Academic Article	Proteasomal degradation of Runx2 shortens parathyroid hormone-induced anti-apoptotic signaling in osteoblasts. A putative explanation for why intermittent administration is needed for bone anabolism.
Academic Article	Glucocorticoids act directly on osteoblasts and osteocytes to induce their apoptosis and reduce bone formation and strength.
Academic Article	Rosiglitazone causes bone loss in mice by suppressing osteoblast differentiation and bone formation.
Academic Article	Quantifying osteoblast and osteocyte apoptosis: challenges and rewards.
Academic Article	Intermittent parathyroid hormone administration counteracts the adverse effects of glucocorticoids on osteoblast and osteocyte viability, bone formation, and strength in mice.
Academic Article	Connexin 43 is required for the anti-apoptotic effect of bisphosphonates on osteocytes and osteoblasts in vivo.
Academic Article	Estrogens attenuate oxidative stress and the differentiation and apoptosis of osteoblasts by DNA-binding-independent actions of the ERalpha.
Academic Article	FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in mice.
Academic Article	Glucocorticoids and tumor necrosis factor a increase oxidative stress and suppress Wnt protein signaling in osteoblasts.
Academic Article	Glucocorticoid-induced osteoporosis and osteonecrosis.
Academic Article	Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.
Academic Article	Control of bone mass and remodeling by PTH receptor signaling in osteocytes.
Academic Article	Suppression of autophagy in osteocytes mimics skeletal aging.
Academic Article	Matrix-embedded cells control osteoclast formation.
Academic Article	Skeletal involution by age-associated oxidative stress and its acceleration by loss of sex steroids.
Academic Article	Estrogen receptor-a signaling in osteoblast progenitors stimulates cortical bone accrual.
Academic Article	The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone.
Academic Article	Intermittent PTH stimulates periosteal bone formation by actions on post-mitotic preosteoblasts.
Academic Article	Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age.
Academic Article	Continuous elevation of PTH increases the number of osteoblasts via both osteoclast-dependent and -independent mechanisms.
Concept	Osteoblasts
Academic Article	An Inverse Relationship between Prevalence of Osteoblast Apoptosis and Rate of Bone Formation with Intermittent, but not Sustained, Elevation of PTH in Mice
Academic Article	Glucocorticoid excess decreases the number of osteoblast progenitors in the marrow and increases osteoblast apoptosis
Academic Article	Estrogen receptor-alpha signaling in osteoblast progenitors stimulates cortical bone accrual
Academic Article	Osteoblast Specific Expression of 11B-HSD2 in Transgenic Mice Abrogates Steroid-Induced Apoptosis and Attenuates Loss of BMD and Strength
Academic Article	Intermittent parathyroid hormone administration prevents glucocorticoid-induced osteoblast and osteocyte apoptosis, decreased bone formation, and reduced bone strength in mice
Academic Article	Glucocorticoids and tumor necrosis factor alpha increase oxidative stress and suppress Wnt protein signaling in osteoblasts
Academic Article	PTH increases bone formation by postponing osteoblast apoptosis, not by increasing precursor proliferation
Academic Article	Increased RANKL Expression and Osteoclast Numbers, but No Change in Osteoblast Number or Apoptosis in a Murine Model of Sustained PTH Elevation: Mechanism of Decreased Bone Density and Strength
Academic Article	The ERa of Osteoblast Progenitors is Required for Normal Accrual of Cortical Bone Mass Independently of Estrogens
Academic Article	An Essential Role for Smad3 in Bone Formation through Regulation of Osteoblast Differentiation
Academic Article	Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates bone porosity with age
Academic Article	Estrogens attenuate oxidative stress and osteoblast differentiation, proliferation and apoptosis by DNA binding-independent actions of the ERa
Academic Article	Activation of PPARy2 by Rosiglitazone Causes Bone Loss Associated with Increased Marrow Adiposity and Decreased Osteoblast Number in Mice
Academic Article	Like Estrogen, Androgen Exert Potent and Direct Anti-apoptotic Effects on Osteoblasts and Osteocytes in vivo and in vitro
Academic Article	A Strong Positive Correlation Between the Prevalence of Osteoblast Apoptosis and the Activation Frequency Across a Wide Range of Bone Remodeling Rates
Academic Article	The spacial and temporal relationships between osteoclasts and osteoblasts during bone remodeling require parallel as well as serial signal pathways
Academic Article	Demonstration of programmed cell death (apoptosis) in osteoblasts: evidence for modulation of this process by growth factors and cytokines
Academic Article	Fox-O-mediated defense against oxidative stress in osteoblasts is indispensable for skelatal homostasis in mice
Academic Article	Suppression of Autophagy in Osteoblasts and Osteocytes Increases Oxidative Stress and Recapitulates the Effects of Aging on the Murine Skeleton
Academic Article	The Effects of Androgens on Murine Cortical Bone Do Not Require AR or ERa Signaling in Osteoblasts and Osteoclasts.
Academic Article	Deletion of Rac in Mature Osteoclasts Causes Osteopetrosis, an Age-Dependent Change in Osteoclast Number, and a Reduced Number of Osteoblasts In Vivo.
Academic Article	Skeletal inflammation and attenuation of Wnt signaling, Wnt ligand expression, and bone formation in atherosclerotic ApoE-null mice.
Academic Article	Old age causes de novo intracortical bone remodeling and porosity in mice.
Academic Article	The Pathophysiological Sequence of Glucocorticoid-Induced Osteonecrosis of the Femoral Head in Male Mice.
Academic Article	Oxidation-specific epitopes restrain bone formation.
Grant	Molecular and Cellular Mechanisms of Osteoporosis
Grant	The Role of Osteocyte Survival in Bone Strength

Search Criteria

Osteoblasts