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Search Results to Robert Weinstein

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Weinstein, Robert

One or more keywords matched the following items that are connected to Weinstein, Robert

Item Type	Name
Academic Article	The skeletal effects of glucocorticoid excess override those of orchidectomy in mice.
Academic Article	Proteasomal degradation of Runx2 shortens parathyroid hormone-induced anti-apoptotic signaling in osteoblasts. A putative explanation for why intermittent administration is needed for bone anabolism.
Academic Article	Glucocorticoids act directly on osteoblasts and osteocytes to induce their apoptosis and reduce bone formation and strength.
Academic Article	Quantifying osteoblast and osteocyte apoptosis: challenges and rewards.
Academic Article	Intermittent parathyroid hormone administration counteracts the adverse effects of glucocorticoids on osteoblast and osteocyte viability, bone formation, and strength in mice.
Academic Article	Connexin 43 is required for the anti-apoptotic effect of bisphosphonates on osteocytes and osteoblasts in vivo.
Academic Article	Estrogens attenuate oxidative stress and the differentiation and apoptosis of osteoblasts by DNA-binding-independent actions of the ERalpha.
Academic Article	FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in mice.
Academic Article	Glucocorticoids and tumor necrosis factor a increase oxidative stress and suppress Wnt protein signaling in osteoblasts.
Academic Article	Osteocyte apoptosis.
Academic Article	Osteoprotegerin prevents glucocorticoid-induced osteocyte apoptosis in mice.
Academic Article	Glucocorticoid-induced osteoporosis and osteonecrosis.
Academic Article	Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.
Academic Article	Giant osteoclast formation and long-term oral bisphosphonate therapy.
Academic Article	The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone.
Academic Article	Intermittent PTH stimulates periosteal bone formation by actions on post-mitotic preosteoblasts.
Academic Article	Osteocyte apoptosis is induced by weightlessness in mice and precedes osteoclast recruitment and bone loss.
Academic Article	Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age.
Academic Article	Glucocorticoid-induced osteonecrosis.
Concept	TNF-Related Apoptosis-Inducing Ligand
Concept	Apoptosis
Academic Article	An Inverse Relationship between Prevalence of Osteoblast Apoptosis and Rate of Bone Formation with Intermittent, but not Sustained, Elevation of PTH in Mice
Academic Article	Glucocorticoid excess decreases the number of osteoblast progenitors in the marrow and increases osteoblast apoptosis
Academic Article	Osteoblast Specific Expression of 11B-HSD2 in Transgenic Mice Abrogates Steroid-Induced Apoptosis and Attenuates Loss of BMD and Strength
Academic Article	Intermittent parathyroid hormone administration prevents glucocorticoid-induced osteoblast and osteocyte apoptosis, decreased bone formation, and reduced bone strength in mice
Academic Article	11B-Hydroxysteriod Dehydrogenase Type 2 (11B-HSD2) Overexpression Prevents Glucocorticoid-Induced Apoptosis of Osteoblastic Cells: A Novel Strategy for Dissecting the Mechanism of Steroid-Induced Osteoporosis
Academic Article	Osteoprotegerin prevents glucocorticoid-induced osteocyte apoptosis, decreased bone interstitial fluid, and reduced strenght in mice( in preparation
Academic Article	PTH increases bone formation by postponing osteoblast apoptosis, not by increasing precursor proliferation
Academic Article	Glucocorticoid excess causes apoptosis of osteocytes in murine cortical bone
Academic Article	Increased RANKL Expression and Osteoclast Numbers, but No Change in Osteoblast Number or Apoptosis in a Murine Model of Sustained PTH Elevation: Mechanism of Decreased Bone Density and Strength
Academic Article	Estrogens attenuate oxidative stress and osteoblast differentiation, proliferation and apoptosis by DNA binding-independent actions of the ERa
Academic Article	A Strong Positive Correlation Between the Prevalence of Osteoblast Apoptosis and the Activation Frequency Across a Wide Range of Bone Remodeling Rates
Academic Article	Apoptosis in Bone Cells
Academic Article	Apoptosis in glucocorticoid-induced bone disease
Academic Article	Demonstration of programmed cell death (apoptosis) in osteoblasts: evidence for modulation of this process by growth factors and cytokines
Academic Article	In: Principles of Bone Biology. John Bilezikian, Lawrence Raisz, T. John Martin, eds
Academic Article	The Pathophysiological Sequence of Glucocorticoid-Induced Osteonecrosis of the Femoral Head in Male Mice.
Grant	Molecular and Cellular Mechanisms of Osteoporosis
Grant	The Role of Osteocyte Survival in Bone Strength
Grant	Bisphosphonate effects on osteoclasts and osteocytes in vitro: mechanisms of inhibition of glucocorticoid-induced apoptosis of osteocytes
Grant	Bisphosphonate effects on osteoclasts and osteocytes in vitro: mechanisms of inhibition of glucocorticoid-induced apoptosis of osteocytes

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Apoptosis