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Sequestration of mutated alpha1-antitrypsin into inclusion bodies is a cell-protective mechanism to maintain endoplasmic reticulum function.

Granell S, Baldini G, Mohammad S, Nicolin V, Narducci P, Storrie B, Baldini G. Sequestration of mutated alpha1-antitrypsin into inclusion bodies is a cell-protective mechanism to maintain endoplasmic reticulum function. Mol Biol Cell. 2008 Feb; 19(2):572-86.

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