Myeloma-derived Dickkopf-1 disrupts Wnt-regulated osteoprotegerin and RANKL production by osteoblasts: a potential mechanism underlying osteolytic bone lesions in multiple myeloma.

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Myeloma-derived Dickkopf-1 disrupts Wnt-regulated osteoprotegerin and RANKL production by osteoblasts: a potential mechanism underlying osteolytic bone lesions in multiple myeloma.

Qiang YW, Chen Y, Stephens O, Brown N, Chen B, Epstein J, Barlogie B, Shaughnessy JD. Myeloma-derived Dickkopf-1 disrupts Wnt-regulated osteoprotegerin and RANKL production by osteoblasts: a potential mechanism underlying osteolytic bone lesions in multiple myeloma. Blood. 2008 Jul 01; 112(1):196-207.

View in: PubMed

subject areas

Animals
Base Sequence
Cell Differentiation
Cell Line
Cell Line, Tumor
Coculture Techniques
DNA Primers
Gene Expression
Gene Silencing
Humans
Intercellular Signaling Peptides and Proteins
Mice
Multiple Myeloma
Osteoblasts
Osteolysis
Osteoprotegerin
RANK Ligand
Recombinant Proteins
Reverse Transcriptase Polymerase Chain Reaction
RNA, Messenger
RNA, Neoplasm
Transfection
Tumor Cells, Cultured
Wnt Proteins
Wnt3 Protein
Wnt3A Protein